BBA - Molecular Basis of Disease

BBA - Molecular Basis of Disease

BBA - Molecular Basis of Disease
External linkMolecular Basis of Multiple Sclerosis
Edited by A. Prat
Volume 1812, Issue 2, Pages 131-282 (February 2011)

With more than 44,200 publications relating to Multiple Sclerosis (MS) during the last 10 years (Pubmed and ISI Thompson, as of October 19th 2010), it has become extremely challenging for students, post-doctoral fellows, academic investigators and clinicians to follow this tremendously active field of research. Furthermore, the annual number of manuscripts listing MS as a keyword or including MS in the abstract has doubled from 2594 in 2000, to 5996 in 2009 (see Table 1). At this pace, it is more difficult to identify, retrieve and analyze important contributions, even in very narrow and sub-specialized fields of MS science.
 

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Dr. Prat

Dr. Prat obtained his B.Sc. degree in biochemistry from Université de Montréal in 1990, an M.Sc. degree in physiology in 1994 and an MD degree from the same university in 1995. Dr. Prat completed his Neurology residency (clinical) training at McGill University (Montreal Neurological Institute) in 2003 after having completed a Ph.D. degree in the laboratory of Dr. Jack P. Antel in 2000. His PhD work focused on the development of an in vitro model of the human Blood-Brain Barrier. Dr. Prat is an active member of the Royal College of Physician and Surgeons of Canada (Neurology). Dr Prat is a staff neurologist at the CHUM-Notre Dame Hospital (Montréal) and as a Professor of Neurology at Université de Montréal in September 2003. Dr. Prat holds the Donald Paty Research Chair of the Multiple Sclerosis Society of Canada and he is a research scholar of the FRSQ. In 2000, he received the S. Weir Mitchell Award of the American Academy of Neurology for his work on Multiple Sclerosis. The current research interests of the Prat lab include the immunological roles of the BBB in Multiple Sclerosis, the mechanisms of monocytes and lymphocyte migration across the BBB and the physiological regulation of the Blood-Brain Barrier functions by glial cells.


  
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