NeuroToxicology - ISSN 0161-813X
Source Normalized Impact per Paper (SNIP): 0.979 Source Normalized Impact per Paper (SNIP):
SNIP measures contextual citation impact by weighting citations based on the total number of citations in a subject field.
SCImago Journal Rank (SJR): 0.933 SCImago Journal Rank (SJR):
SJR is a prestige metric based on the idea that not all citations are the same. SJR uses a similar algorithm as the Google page rank; it provides a quantitative and a qualitative measure of the journal’s impact.
Impact Factor: 3.105 (2019) Impact Factor:
The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years.
© 2017 Journal Citation Reports ® (Clarivate Analytics, 2017)
5 Year Impact Factor: 3.43 (2019) Five-Year Impact Factor:
To calculate the five year Impact Factor, citations are counted in 2016 to the previous five years and divided by the source items published in the previous five years.
© 2017 Journal Citation Reports ® (Clarivate Analytics, 2017)
Volumes: Volume 6
Issues: 6 issues
ISSN: 0161813X

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NeuroToxicology specializes in publishing peer-reviewed original research papers describing the effects of toxic substances on the nervous system across the lifespan as determined in humans and/or experimental models (in vivo, in vitro, in silico). The Journal welcomes papers dealing with the neurotoxic effects of occupationally and environmentally relevant exposures to agents (chemical, physical, biological, pharmacological or naturally occurring), singly or in mixtures, including complex mixtures, such as air pollution. Papers describing neurotoxic outcomes associated with natural disasters, industrial accidents, and terrorist attacks are also welcome.

Experimental (animal, in vitro, in silico) papers focused on the neurotoxic effects of undefined commercial formulas (i.e., pesticide formulations) will be considered only if the authors report the chemical composition of the formulation and/or determine whether neurotoxic effects are due to the active chemical ingredient(s), carrier, or combination. For human studies, the components of formulations or other mixtures should be identified, but if not available, the source of exposure (i.e. commercial formulation, air pollution, wildfires, hurricanes, and other natural or industrial disasters) should be described as fully as possible.

NeuroToxicology welcomes papers describing interventions for mitigating or reversing neurotoxic outcomes, but will accept papers reporting on neuroprotective or neurorestorative properties of formulations, botanical extracts, or other natural products only if full chemical identification and purification information of the active molecule(s) is provided.

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