Secure CheckoutPersonal information is secured with SSL technology.
Free ShippingFree global shipping
No minimum order.
Preface: Endoplasmic reticulum in health and disease
Lorenzo Galluzzi and Oliver Kepp
1. Endoplasmic reticulum stress in the cellular release of damage-associated molecular patterns
Alejandra Wu Chuang, Oliver Kepp, Guido Kroemer and Lucillia Bezu
2. Emerging roles of the unfolded protein response (UPR) in the nervous system: A link with adaptive behavior to environmental stress?
Mei-Li Díaz-Hung, Gabriela Martínez and Claudio Hetz
3. Type I interferons and endoplasmic reticulum stress in health and disease
Jenny Sprooten and Abhishek D. Garg
4. The role of mitochondria-associated membranes in cellular homeostasis and diseases
Mariasole Perrone, Natascia Caroccia, Ilaria Genovese, Sonia Missiroli, Lorenzo Modesti, Gaia Pedriali, Bianca Vezzani, Veronica Angela Maria Vitto, Michele Antenori, Magdalena Lebiedzinska-Arciszewska, Mariusz R. Wieckowski, Carlotta Giorgi and Paolo Pinton
5. Sarcoplasmic reticulum and calcium signaling in muscle cells: Homeostasis and disease
Roberto Bravo-Sagua, Valentina Parra, Felipe Muñoz-Cordova, Pablo Sanchez-Aguilera, Valeria Garrido, Ariel Contreras-Ferrat, Mario Chiong and Sergio Lavandero
6. Endoplasmic reticulum in viral infection
7. Targeting endoplasmic reticulum stress and autophagy as therapeutic approaches for neurological diseases
Annadurai Thangaraj, Susmita Sil, Ashutosh Tripathi, Ernest T. Chivero, Palsamy Periyasamy and Shilpa Buch
International Review of Cell and Molecular Biology, Volume 350, covers all aspects of endoplasmic reticulum (ER) biology. With its multiple cellular functions, including ion storage as well protein folding, trafficking and secretion, the regulation of homeostasis within the ER is crucial to organismal health. New sections in this updated volume include DAMP emission upon ER stress, Protein misfolding disordersm Type I interferon response and ER stress, ER and autophagosome biogenesis, Mitochondria-associated membranes, ER calcium signaling in excitable cells, and ER in viral infections.
- Summarizes endoplasmic reticulum-related pathologies
- Gives an update on the immunological aspects of ER-stress
- Brings the links between ER homeostasis and autophagy
Cell biologists, Immunologists, Oncologists
- No. of pages:
- © Academic Press 2020
- 2nd March 2020
- Academic Press
- Hardcover ISBN:
- eBook ISBN:
Lorenzo Galluzzi is Assistant Professor of Cell Biology in Radiation Oncology at the Department of Radiation Oncology of the Weill Cornell Medical College, Honorary Assistant Professor Adjunct with the Department of Dermatology of the Yale School of Medicine, Honorary Associate Professor with the Faculty of Medicine of the University of Paris, and Faculty Member with the Graduate School of Biomedical Sciences and Biotechnology of the University of Ferrara, the Graduate School of Pharmacological Sciences of the University of Padova, and the Graduate School of Network Oncology and Precision Medicine of the University of Rome “La Sapienza”. Moreover, he is Associate Director of the European Academy for Tumor Immunology and Founding Member of the European Research Institute for Integrated Cellular Pathology. Galluzzi is best known for major experimental and conceptual contributions to the fields of cell death, autophagy, tumor metabolism and tumor immunology. He has published over 450 articles in international peer-reviewed journals and is the Editor-in-Chief of four journals: OncoImmunology (which he co-founded in 2011), International Review of Cell and Molecular Biology, Methods in Cell biology, and Molecular and Cellular Oncology (which he co-founded in 2013). Additionally, he serves as Founding Editor for Microbial Cell and Cell Stress, and Associate Editor for Cell Death and Disease, Pharmacological Research and iScience.
Assistant Professor of Cell Biology in Radiation Oncology, Department of Radiation Oncology, Weill Cornell Medical College, NY, USA
Elsevier.com visitor survey
We are always looking for ways to improve customer experience on Elsevier.com.
We would like to ask you for a moment of your time to fill in a short questionnaire, at the end of your visit.
If you decide to participate, a new browser tab will open so you can complete the survey after you have completed your visit to this website.
Thanks in advance for your time.