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A Study Guide to Anesthesia, 5th Edition, and Basics of Anesthesia, 4th Edition
To order this title, and for more information, click here
By
Lorraine Sdrales, MD, Assistant Clinical Professor, Department of Anesthesia and Perioperative Care, University of California, San Francisco, School of
Medicine, San Francisco, CA; Staff Anesthesiologist, Cedars-Sinai Medical Center, Los Angeles, CA
Ronald Miller, MD, Professor and Chairman, Department of Anesthesia and Perioperative Care, Professor of Cellular and Molecular Pharmacology, University
of California, San Francisco, School of Medicine, San Francisco, CA
Robert Stoelting, MD, Professor and Chairman, Department of Anesthesia, Indiana University School of Medicine, Indianapolis, IN
Description
Hundreds of challenging review questions cover a complete range of essential topics in anesthesia -- from physiological and pharmacologic
principles through anesthetic machine systems, anesthetic delivery in a variety of settings, and anesthesia administration for a full
range of disease states. Chapters progress from basic to advanced topics, making it easy to assess any knowledge level. The end of each
chapter has complete answers as well as specific page references to Miller: Anesthesia , 5th Edition and Stoelting & Miller: Basics of
Anesthesia, 4th Edition.
Audience
Students, residents and practioners in anesthesia.
Contents
BASICS PHARMACOLOGIC PRINCIPLES: What Does Pharmacokinetics Describe?. What Does Pharmacodynamics Describe?. What Is the Role of Receptors
in Drug Pharmacology? TERMINOLOGY and DEFINITIONS: What Are Agonist Drugs?. What Are Antagonist Drugs?. Define Competitive and Noncompetitive
Antagonism: Which of These Is Overcome By Higher Concentrations of the Drug?. What Does an Additive Drug Effect Mean?: Give an Example.
What Does a Synergistic Drug Effect Mean?: Give an Example. When an Individual Is Referred to as Hyporeactive to a Drug, What Does It
Mean? Hyperractive?. What Is Drug Tolerance?. What Is Drug Cross-Tolerance?. What Does Tachyphylaxis to a Drug Refer To?. How Do the
Tissues of the Body Equilibrate with the Partial Pressure of the Inhaled Anesthetic?: Write the Equilibrium Equation for Equilibration
of PI to Pbr. What Does the Alveolar Partial Pressure (PA) Reflect?: What Are Some Ways in Which It Is Used Clinically?. What Are the
Six Factors that Act Simultaneously to Determine the PA?: How Much Does Metabolism and Percutaneous Loss of Inhaled Anesthetics Influence
the PA. Of the Six Factors Listed Above, Which Three Determine the Inputof the Inhaled Anesthetic?. Of the Six Factors Listed Above,
Which Three Determine the Uptake of the Inhaled Anesthetic?. How Much Does Metabolism and Percutaneous Loss of Inhaled Anesthetics Influence
the PA?. What Is the Concentration Effect? Clinically, With Which Inhaled Agent Is the Concentration Effect Solely Possible? Why? /With
Time, Should the PI Be Increased, Decreased, Or Kept Constant? Why?. What Would Happen if the PI Were Maintained Constant?. What Is
the Second Gas Effect? Does This Depend on or Occur Independent of the Concentration Effect?. Give an Exmple of the Second Gas Effect.
What Is Alveolar Hyperoxygenation? By What Percent Does the Pao2 Increase During Alveolar Hyperoxygenation?. Is the Second Gas Effect
Clinically Significant?. How Does Increasing Alveolar Ventilation (VA) Affect the Induction of Inhaled Anesthesia?. What Is an Idiosyncratic
Reaction to a Drug? What Does It Most Commonly Result From?. What Does a Dose-Response Curve Illustrate? Draw and Label One. How Is
the Potency of a Drug Depicted on a Dose Response Curve?. What Does ED 50 Mean?. What Would Move the Curve to the Left? To the Right?.
What Influences the Slope of a Dose-Response Curve?. What Does It Mean When the Curve Is Steep? What Is the Potential Clinical Problem
Associated with a Steep Curve?. What Is a Drug's Efficacy? How Is It Depicted on the Dose-Response Curve?. What May Limit Drug Efficacy
Clinically?. How Does Individual Variability Alter the Dose-Response Curve? PHARMACOKETICS of INHALED ANESTHETICS: What Does the Pharmacokinetics
of Inhaled Anesthetics Describe? Where Is the Principal Site of Metabolism of Inhaled Anesthetics?. What Is the Objective of Inhalation
Anesthesia?. How Does Controlling the Inspired Partial Pressure (PI) of an Inhaled Anesthetic Influence the Onset of Anesthesia?. How
Do the Tissues of the Body Equilibrate with the Partial Pressure of the Inhaled Anesthetic? Write the Equilibrium Equation for Equilibration
of PI to Pbr. What Does the Alveolar Partial Pressure (PA) Reflect? What Are Some Ways in Which It Is Used Clinically?. What Are the
Six Factors that Act Simultaneously to Determine the PA? How Much Does Metabolism and Percutaneous Loss of Inhaled Anesthetics Influence
the PA?. Of the Six Factors Listed Above, Which Three Determine the Input of the Inhaled Anesthetic?. Of the Six Factors Listed Above,
Which Three Determine the Uptake of the Inhaled Anesthetic?. How Much Does Metabolism and Percutaneous Loss of Inhaled Anesthetics Influence
the PA?. What Is the Concentration Effect? Clinically, With Which Inhaled Agent Is the Concentration Effect Solely Possible? Why?. With
Time, Should the PI Be Increased, Decreased, Or Kept Constant? Why?. What Would Happen if the PI W Ere Maintained Constant?. What Is
the Second Gas Effect? Does This Depend on or Occur Independent of the Concentration Effect?. Give an Exmple of the Second Gas Effect.
What Is Alveolar Hyperoxygenation? By What Percent Does the Pa02 Increase During Alveolar Hyperoxygenation?. Is the Second Gas Effect
Clinically Significant?. How Does Increasing Alveolar Ventilation (VA) Affec the Induction of Inhaled Anesthesia? ANSWERS
| Bibliographic details |
Paperback, 592 pages, publication date: MAR-2001
ISBN-13: 978-0-443-07978-8
ISBN-10: 0-443-07978-1
Imprint: CHURCHILL LIVINGSTONE
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Last update: 10 Sep 2009
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