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 A section of Mutation Research - only available as part of a subscription to Mutation Research/Full Set
NOW PUBLISHED
Special issue on
Nutrigenomics
Edited by L.R. Ferguson, A.N. Shelling, D. Lauren, J.A. Heyes, W.C. McNabb
Editors:
P.J. Stambrook, L.H.F. Mullenders, L.R. Ferguson
See editorial board for all editors information
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Announcement
Message of gratitude from the Editors to our 2007 reviewers
Description
The scope of Mutation Research: Molecular and Fundamental Mechanisms broadly encompasses all aspects of research that address the
detection of mutations, the mechanisms by which mutations in genes and chromosomes arise, and the modulation of mutagenesis by mutation
avoidance pathways such as DNA repair, cell cycle control and apoptosis. It includes the role of genetic variation in the genesis and
manifestation of mutations, ranging from the variable manner in which xenobiotics are metabolized to variations in the capacity of cells
to replicate and repair damaged DNA. It also includes the contributions of these mechanisms, when perturbed, to animal disease models
and to human disease, with particular emphasis on carcinogenic mechanisms. Chromosome stability is paramount for maintaining cellular
homeostasis. Therefore, the Journal will publish articles on the genesis of aneuploidy and isodisomy, including the roles played by cell
cycle checkpoints, spindle microtubules, centrosomes and kinetocore proteins, and agents that might disrupt them. Since isodisomy can
occur as a consequence of recombination, all aspects of homologous recombination and non-homologous end joining are appropriate. Submission
of appropriate epidemiological studies as well as consequences, including methods for high throughput SNP detection, DNA microarrays
and proteomic approaches, are welcome. The broader scope of the journal is a reflection of the rapid advances in the field of mutation
research and the recognition that cellular responses to DNA damage, including cell cycle checkpoint arrest and apoptosis, cannot be dissociated
from the immediate mechanisms by which DNA is damaged and repaired.
| Bibliographic details |
ISSN: 0027-5107
Imprint: ELSEVIER
Commenced publication 1964
Subscriptions for the year 2009,
Volumes 660-671,
24 issues |
| Price and Ordering |
Personal price:
EUR 323 for European countries and Iran
JPY 49,800 for Japan
USD 434 for all countries except Europe, Japan and Iran
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Institutional price:
USD 7,101 for all countries except Europe, Japan and Iran
EUR 6,345 for European countries and Iran
JPY 840,100 for Japan
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Conditions of sale & ordering procedures, and links to our regional sales offices.
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For an overview of recently-dispatched issues, see the Journal issue
dispatch dates
Audience
Mutageneticists, General Toxicologists, Molecular Geneticists, Cancer Researchers, Radiobiologists.
Impact factor of this journal
2007: 4.159
© Journal Citation Reports 2008, published by Thomson Reuters
602/177
Last update: 9 Oct 2008
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